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Aqueous gentian violet, 1-2%: apply to infected area with swab.
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Evaluation/outcome criteria:
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Oral mucosa intact, lesions healed, no evidence of infection.
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Feeds well; maintains weight or regains weight lost, if any.
-
Neonatal sepsis
-
Pathophysiology—generalized infection; may overwhelm infant’s immature immune system.
-
Etiology:
-
Prolonged rupture of membranes.
-
Long, difficult labor.
-
Resuscitation procedures.
-
Maternal infection (e.g., beta-hemolytic streptococcus vaginosis).
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Aspiration—amniotic fluid, formula, mucus.
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Iatrogenic (nosocomial)—caused by infected health personnel or equipment.
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Assessment:
-
Respirations—irregular, periods of apnea.
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Irritability or lethargy.
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Analysis/nursing diagnosis:
-
Fatigue related to increased oxygen needs.
-
High risk for infection related to septic condition.
-
Nursing care plan/implementation:
-
Cultures (spinal, urine, blood).
-
Check vitals.
-
Monitor respirations.
-
Give medications, as ordered.
-
Evaluation/outcome criteria:
-
Responds to medical/nursing regimen (all assessment findings within normal limits).
-
Parent(s) verbalize understanding of diagnosis, treatment; demonstrate appropriate techniques in participating in care (as possible).
-
Parent(s) demonstrate effective coping with situation; express satisfaction with care.
-
Disorders affecting nutrition: infant of the diabetic mother (IDM)
-
Pathophysiology—hyperplasia of pancreatic beta cells —> increased insulin production —» excessive deposition of glycogen in muscles, subcutaneous fat, and tissue growth. Results in fetal:
- Macrosomia—LGA infant.
-
Enlarged internal organs—common.
-
Cardiomegaly.
-
Hepatomegaly.
-
Splenomegaly.
-
Neonatal—inadequate carbohydrate reserve to meet energy needs.
-
Associated with increased incidence of:
-
Congenital anomalies (five times average incidence with pregestational diabetes) includes cardiac, pelvic, and spinal anomalies.
-
Preterm birth: respiratory distress syndrome (RDS); increased insulin needs prenatally lead to decreased surfactant production.
-
Fetal dystocia—due to CPD.
-
Neonatal metabolic problems:
-
Hypoglycemia.
-
Hypocalcemic tetany.
-
Metabolic acidosis.
-
Hyperbilirubinemia.
-
Etiology—high circulating maternal glucose levels during fetal growth and development; loss of maternal glucose supply following birth; decreased hepatic gluconeogenesis.
-
Assessment:
-
Characteristics of IDM.
-
Hypoglycemia—Dextrostix or Chem-strip to heel stick at:
-
30 min X 2.
-
1,2, and 4 hr of age; before meals X 4 or until stable.
-
Chemstrip: if <20 mg/dl, must draw glucose stat.
-
Hypoglycemia laboratory values for term infant: under 40 mg/dl.
-
Hypoglycemia laboratory values for preterm infant: under 40 mg/dl.
-
Behavioral signs—tremors; twitching, hypotonia, seizures.
-
Gestational age, since macrosomia may mask prematurity.
-
Hypocalcemia—usually within first 24 hr
-
Irritability.
-
Coarse tremors, twitching, convulsions.
-
Birth injuries.
-
Fractures: clavicle, humerus, skull.
-
Brachial palsy.
-
Intracranial hemorrhage/signs of increased intracranial pressure.
-
Cephalohematoma.
-
Respiratory distress.
-
Nasal flaring.
-
Sternal retraction.
-
Intercostal retractions.
-
Cyanosis—central.
-
Expiratory grunt.
-
Jaundice.
-
Analysis/nursing diagnosis:
-
High risk for injury related to CPD, dystocia.
-
Altered cardiopulmonary tissue perfusion related to placental insufficiency, RDS.
-
Impaired gas exchange related to RDS.
-
Altered nutrition, less than body requirements, related to hypoglycemia, hypocalcemia.