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Complications affecting sensory/perceptual functions:
pregnancy-induced hypertension (PIH); preedampsia/eclampsia
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Pathophysiology
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Generalized arteriospasm —> increased peripheral resistance, decreased tissue
perfusion, and hypertension.
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Kidney:
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Reduced renal perfusion and vasospasm —> glomerular lesions.
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Damage to membrane —> loss of serum protein (albuminuria). Note: Reduced serum
albumin/globulin (A/G) ratio alters blood osmolarity —> edema.
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Increased tubular reabsorption of sodium —> increased water retention
(edema).
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Release of angiotensin contributes to vasospasm and hypertension.
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Brain: decreased oxygenation, cerebral edema, and vasospasm —> visual disturbances and
hyperirritability, convulsions, and coma.
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Uterus: decreased placental perfusion —> increased risk of SGA baby, abruptio placentae,
oligohydramnios.
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Etiology: unknown.
Risk factors:
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Pregnancy - occurs only when a functioning trophoblast is present; more common in
first pregnancies;
develops after week 20 of gestation.
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Coexisting conditions - diabetes, multifetal gestation, polyhydramnios, renal
disease.
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Angiotensin gene T235.
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Assessment - types:
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Preeclampsia - mild
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Hypertension - systolic increase of 30 mm Hg or more over baseline; diastolic
rise of 15 mm Hg or more.
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Proteinuria - 1 g/day.
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Edema - digital and periorbital; weight gain over 0.45 kg (1 lb)/wk.
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Preeclampsia - severe
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Increasing hypertension - systolic at or above 160 mm Hg or more than 50 mm Hg over
baseline; diastolic, 110 mm Hg or more
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Urine: proteinuria (5 grams or more in 24 hours); oliguria (400 mL or less in 24 hours).
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Hemoconcentration, hypoproteinemia, hypernatremia, hypovolemic condition.
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Nausea and vomiting.
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Epigastric pain - due to edema of liver capsule.
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Cerebral or visual disturbances (before convulsive state):
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Disorientation and somnolence.
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Severe frontal headache.
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Increased irritability; hyperreflexia.
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Blurred vision, halo vision, dimness, blind spots.
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HELLP Syndrome (Hemolysis, Elevated Liver enzymes, and Low Platelets).
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Eclampsia
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Tonic and clonic convulsions; coma.
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Renal shutdown - oliguria, anuria.
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Assessment - hospitalized woman:
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Vital signs (blood pressure in side-lying position, pulse, respirations) - q2-4h, while
awake (if mild to moderate preeclampsia) or as necessary. Note: Record, report persistent hypertension.
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Fetal heart tones at time of vital signs.
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Deep-tendon reflexes (DTR) and clonus - to identify/monitor CNS
hyperirritability.
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I&O - to identify diuresis. (Note: oliguria indicates pathologic
progression.)
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Urinalysis (clean-catch) for protein, daily or after each voiding, as
necessary.
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Signs of pathologic progression.
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Signs of labor, abruptio placentae (Note: high blood pressure, or a rapid drop,
may initiate abruptio), DIC.
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Emotional status.
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Daily weight, amount/distribution of edema (pitting; pedal, digital,
periorbital)-to identify signs of mobilization of tissue fluid, diuresis.
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Analysis/nursing diagnosis:
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Fluid volume
excess: hemoconcentration, edema related to altered blood osmolarity and
sodium/water retention.
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Altered nutrition» less than
body requirements: protein deficiency related to loss through damaged
renal membrane.
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Altered tissue
perfusion related to increased peripheral resistance and vasospasm in
renal, cardiovascular system.
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Altered urinary
elimination: oliguria, anuria related to hypovolemia, vasospasm.
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Sensory/perceptual
alterations: visual disturbances, hyperirritability related to cerebral
edema, decreased oxygenation to brain.
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Anxiety related to symptoms, implications of pathophysiology.
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Diversional activity
deficit related to need for reduced environmental stimuli, bed
rest.
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Risk for injury related to seizure.
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Prognosis:
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Good - symptoms mild, respond to treatment.
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Poor - convulsions (number and duration); persistent coma; hyperthermia, tachycardia (120
beats/min); cyanosis, and liver damage.
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Terminal - pulmonary edema, congestive heart failure, acute renal failure, cerebral hemorrhage.
The earlier the symptoms appear, the poorer the outcome for the pregnancy.
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Nursing care plan/implementation: Goal: health teaching.
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Rest - frequent naps in lateral Sims position.
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Immediate report of danger
signs:
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Digital and periorbital edema.
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Severe headache, irritability.
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Visual disturbances.
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Epigastric pain.
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Do
roll-over test (blood pressure while on back and lateral
positions).
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Importance of regular prenatal visits.
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Monitoring own blood pressure between prenatal visits.
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Nursing care plan/implementation - hospitalized
woman:
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Goal: reduce environmental
stimuli to minimize stimulation of hyperirritable CNS. Limit visitors
and phone calls.
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Goal: emotional
support.
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Encourage verbalization of anxiety, fears, concerns.
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Explain all procedures, seizure precautions.
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Goal: supportive
care.
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Encourage bed rest - to increase tissue perfusion, promote diuresis.
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Position: lateral Sims’ - to reduce risk of supine hypotensive syndrome.
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Goal: health
teaching. Rest with reduced stimuli.
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Goal: monitor and
administer drugs as ordered.
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Anticonvulsants (especially magnesium sulfate).
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Antihypertensives.
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Diuretics (used rarely, and only in presence of congestive heart failure
[CHF]).
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Blood volume expanders.
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Goal: seizure
precautions. To safeguard mater-nal/fetal status:
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Observe for signs and symptoms of impending convulsion:
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Frontal headache.
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Epigastric pain.
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Sharp cry.
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Eyes fixed; unresponsive.
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Facial twitching.
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Emergency items (suction equipment, airway, drugs, IV fluids) immediately
available.
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Goal: convulsion
care (woman with eclampsia).
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Maintain patent airway; administer oxygen.
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Safety - padded bed rails.
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Reduce environmental stimuli: dim lights, quiet.
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Observe, report, and record:
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Onset and progression of convulsion.
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If followed by coma or incontinence.
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Prepare for immediate cesarean delivery; check FHR. Close observation for 48 hours
postpartum, even if no further convulsions (or no convulsions to date).
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Evaluation/outcome criteria:
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Woman complies with medical/nursing plan of care.
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Woman’s symptoms respond to treatment; progression halted.
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Woman carries uneventful pregnancy to successful termination.