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Disorders affecting protective functions:
postpartal infection (Table 4-13).
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General aspects
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Definition—reproductive system infection occurring during the postpartal perio
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Pathophysiology—
bacterial invasion of birth canal; most common: localized infection of the lining of the uterus (endometritis).
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Etiology:
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Anaerobic nonhemolytic streptococci.
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coli
- trachomatis (bacteroides).
- Staphylococci
- Predisposing conditions:
- Anemi
- PROM.
- Prolonged labor.
- Repeated vaginal examinations during labor.
- Intrauterine manipulation—e.g., manual extraction of placent
- Retained placental fragments.
- Postpartum hemorrhag
- Assessment:
- Fever 38°C (100.4°F) or more on two or more occasions, after first 24 hr postpartum.
- Other signs of infection: pain, malaise, dysuria, subinvolution, foul lochial odor.
- Analysis/nursing diagnosis:
- Fluid volume deficit related to excessive blood loss, anemi
- Knowledge deficit related to danger signs of postpartum perio
- High risk for injury related to infection.
- Nursing care plan/implementation: prevention
- Goal: prevent anemia.
- Minimize blood loss - accurate postpartal assessment and management of bleeding.
- Diet: high protein, high vitamin.
- Vitamins, iron - suggest continuing prenatal pattern until postpartum checkup.
- Goal: prevent entrance/transport of microorganisms.
Strict aseptic technique during labor, birth, and postpartum (standard precautions).
- Minimize vaginal examinations during labor.
- Perineal care.
- Goal: health teaching.
- Hand washing - before and after each pad change, after voiding or defecatin
- Perineal care - from front to back; use clear, warm water or mild antiseptic solution as a cascade; do not separate labi
- Maintain sterility of pads; apply from front to back.
- Avoid use of tampons until normal menstrual cycle resumes.
- Evaluation/outcome criteria:
- Woman has assessment findings within normal limits:
- Vital signs.
- Rate of involution (fundal height, consistency).
- Lochia: character, amount, odor.
- Woman avoids infection.
- Endometritis— infection of lining of uterus.
- Pathophysiology— see I General aspects, p. 15
- Etiology— most common: invasion by normal body flora (, anaerobic streptococci).
- Characteristics:
- Mild, localized—asymptomatic, or lowgrade fever.
- Severe—may lead to ascending infection, parametritis, pelvic abscess, pelvic thrombophlebitis.
- If remains localized, self-limiting; usually resolves within 10 days.
- Assessment:
- Signs of infection: fever, chills, malaise, anorexia, headache, backach
- Uterus: large, boggy, extremely tender.
- Subinvolution.
- Lochia: dark brown; foul odor.
- Analysis/nursing diagnosis:
- Anxiety/fear related to effects on self and newborn.
- Self-esteem disturbance and altered role performance related to inability to meet own expectations regarding parenting, secondary to unexpected hospitalization.
- Pain related to inflammation/infection.
- Ineffective individual coping related to physical discomfort and psychological stress associated with self-concept disturbance; worry, guilt, concern regarding newborn at hom
- Altered family processes— interruption of adjustment to altered life pattern related to postpartal infection/hospitalization.
- Nursing care plan/implementation:
- Goal: prevent cross-contamination. Contact-item isolation.
- B Goal: facilitate drainag Position: semiFowler’s.
- Goal: nutrition/hydration.
- (l) Diet: high-calorie, high-protein, highvitamin.
- Push fluids to 4000 mL/day (oral or IV, or both, as ordered).
- I&O.
- Goal: increase uterine tone/facilitate involution. Administer medications, as ordered (, oxytocics, antibiotics).
- Goal: minimize energy expenditure, as possibl
- Bed rest.
- Maximize rest, comfort.
- Goal: emotional support.
- Encourage verbalization of anxiety, concerns.
- Keep informed of progress.
- Evaluation/outcome criteria:
- Vital signs stable, within normal limits.
- All assessment findings within normal limits.
- Unable to recover organism from discharge.
- Urinary tract infections
- Pathophysiology - normal physiological changes associated with pregnancy (, ureteral dilation) and the postpartal period (, diuresis, increased bladder capacity with diminished sensitivity of stretch receptors) —> increased susceptibility to bacterial invasion and growth —-» ascending infections (cystitis, pyelonephritis).
- Etiology: usually bacterial.
- Predisposing factors:
- Birth trauma to bladder, urethra, or meatus.
- Bladder hypotonia with retention (due to intrapartal anesthesia or trauma).
- Repeated or prolonged catheterization, or poor technique
- Weakening of immune response secondary to anemia, hemorrhage
- Assessment:
- Maternal vital signs (fever, tachycardia).
- Dysuria, frequency (flank pain—with pyelonephritis).
- Feeling of “not emptying” bladder.
- Cloudy urine; frank pus.
- Analysis/nursing diagnosis:
- Altered urinary elimination related to diuresis, dysuria, inflammation/infection.
- Pain related to dysuria secondary to cystitis, Knowledge deficit related to self-care (perineal care).
- Nursing care plan/implementation:
- Goal: minimize perineal edem Perineal ice pack in fourth stage—to limit swelling secondary to trauma, facilitate voidin
- Goal: prevent overdistention of bladder.
- Monitor level of fundus, lochia, bladder distention. (Note: Distended bladder displaces uterus, limits its ability to contract —> boggy fundus, increases its vaginal bleedin)
- Encourage fluids and voiding; I&O.
- Aseptic technique for catheterization.
- Slow emptying of bladder on catheterization—to maintain ton
- Goal: identification of causative organism— to facilitate appropriate medication (antibiotics). Obtain clean-catch (or catheterized) specimen for culture and sensitivity.
- Goal: health teachin See previous discussion of fluids, general hygiene, diet, and medications.
- Evaluation/outcome criteria:
- Voiding: quantity sufficient (although Urine character: clear, amber, or straw colore
- Vital signs: within normal limits.
- No complaints of frequency, urgency, burning on urination, flank pain.
- Mastitis - inflammation of breast tissue:
- Pathophysiology - local inflammatory response to bacterial invasion; suppuration may occur; organism can be recovered from breast milk.
- Etiology - most common: Staphylococcus aureus; source - most common: infant’s nose, throat.
- Assessment:
- Signs of infection (may occur several weeks: postpartum).
- Fever.
- Chills.
- Tachycardi
- Malais
- Abdominal pain.
- Breast
- Reddened area(s).
- Localized/generalized swellin
- Heat, tenderness, palpable mass.
- Analysis/nursing diagnosis:
- Impaired skin integrity related to nipple fissures, cracks.
- Pain related to tender, inflamed tissue secondary to infection.
- Disturbance in body image, self-esteem related to association of breastfeeding with female identity and rol
- Anxiety/fear related to sexuality; impact on breastfeeding, if any.
- Nursing care plan/implementation:
- Goal: prevent infection. Health teaching in early postpartum:
- Hand washin
- Breast care - wash with warm water only (no soap) - to prevent removing protective body oils.
- Let breast milk dry on nipples to prevent drying of tissu
- Clean bra (with no plastic pads or liners) to support breasts, reduce friction, minimize exposure to microorganisms.
- Good breastfeeding techniques (see p. 157).
- Alternate position of infant for nursing to change pressure areas.
- Goal: comfort measures.
- Encourage bra or binder - to support breasts, reduce pain from motion.
- Local heat or ice packs as ordered - to reduce engorgement, pain.
- Administer analgesics, as necessary.
- Goal: emotional support.
- Encourage verbalization of feelings, concerns.
- If breastfeeding is discontinued, reassure woman she will be able to resume breastfeeding
- Goal: promote healing.
- Maintain lactation (if desired) by manual expression or breast pump, q4h.
- Administer antibiotics as ordered
- Evaluation/outcome criteria:
- Woman promptly responds to medical/ nursing regimen.
- Symptoms subsid
- Assessment findings within normal limits.
- Woman successfully returns to breastfeedin
- Thrombophlebitis
- Pathophysiology— inflammation of a vein secondary to lodging of a clot.
- Etiology:
- Extension of endometritis with involvement of pelvic and femoral veins.
- Clot formation in pelvic veins following cesarean birt
- Clot formation in femoral (or other) veins secondary to poor circulation, compression, and venous stasis.
- Assessment:
- Pelvic—pain: abdominal or pelvic tenderness.
- Calf—pain: positive Homans' sign (pain elicited by flexion of foot with knee extended).
- Femoral
- Pain.
- Malaise, fever, chills.
- Swelling—“milk le”
- Analysis/nursing diagnosis:
- Pain in affected region related to local inflammatory respons
- Anxiety/fear related to outcom
- Ineffective individual coping related to unexpected postpartum complications, hospitalization, separation from newborn.
- Impaired physical mobility related to imposed bed rest to prevent emboli formation and dislodging clot (embolus).
- Nursing care plan/implementation:
- Goal: prevent clot formation.
- Encourage early ambulation.
- Position: avoid prolonged compression of popliteal space, use of knee gatc
- Apply thromboembolic disease (TED) hose, as ordered, preoperatively or postoperatively, or both, for cesarean birt
- Goal: reduce threat of embol
- Bed rest, with cradle to support beddin
- Discourage massaging “leg cramps.”
- Goal: prevent further clot formation. Administer anticoagulants, as ordere
- Goal: prevent infection.
- Administer antibiotics, as ordere
- Push fluids.
- Goal: facilitate clot resolution. Heat therapy, as ordere
- Evaluation/outcome criteria:
- Symptoms subside; all assessment findings within normal limits.
- No evidence of further clot formation.